Immobilization of the Erupting Molar in the Syrian Hamster
نویسنده
چکیده
The eruption of teeth, like many growth processes, seems to be an orderly and patterned sequence of events. The speed and frequency with which this process occurs make it ideal for studies in development and growth. Yet the interplay of growing tissues that results in eruption, although important, is still a problem. A number of theories have been proposed that attempt to explain the eruptive movements of teeth and the maturation of supporting tissues. The first, and perhaps most obvious, idea has considered simple root elongation an eruptive force, the root lengthening and propelling the crown occlusally as if it were on stilts. Clinical observations, however, do not support this theory. Massler and Schour' have observed that teeth often erupt even though there has been a failure in root elongation. Furthermore, the distance traveled by some teeth (cuspids) is much greater than the total root length. Experimental evidence has also opposed the root-elongation theory. Gowgiel2"3 prevented root formation in monkeys with irradiation and found that eruption of the crowns proceeded in its normal course. Schour and Van Dyke4 removed the pituitary glands of white rats and observed that, even though eruption was markedly retarded, a root "elongation" did continue in the form of a hard-tissue proliferation at the apex. As an innovation of the root-elongation theory, Sicher5 has proposed that pulpal proliferation or vascularity is a primary factor in eruption. He believed that the expansion of pulpal tissue pressing against the stable fUndic bone and hammock ligament pushes the tooth from its socket. Hertzberg
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تاریخ انتشار 2005